TL;DR
This paper explores how a deficiency in SynCAM1 in certain brain cells contributes to cognitive issues in newborns exposed to sevoflurane.
Contribution
It identifies SynCAM1's role in cognitive impairment linked to anesthetic exposure in neonates.
Findings
SynCAM1 deficiency in parvalbumin interneurons is linked to cognitive impairment.
Sevoflurane exposure exacerbates this issue in neonatal hippocampus.
The study highlights a potential target for mitigating anesthetic-related cognitive effects.
Abstract
The cover image is based on the Original Article SynCAM1 deficiency in the hippocampal parvalbumin interneurons contributes to sevoflurane‐induced cognitive impairment in neonatal by Ming‐ming Zhao et al., https://doi.org/10.1111/cns.14554.
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Taxonomy
TopicsAnesthesia and Neurotoxicity Research