# The language faculty that wasn't: a usage-based account of natural language recursion

**Authors:** Morten H. Christiansen, Nick Chater

PMC · DOI: 10.3389/fpsyg.2015.01182 · 2015-08-27

## TL;DR

This paper challenges the idea of a specialized language faculty, suggesting that recursion in language arises from general sequence learning abilities.

## Contribution

It proposes a usage-based account of recursion, rejecting the need for a dedicated language faculty.

## Key findings

- Evidence from genetics and neuroscience supports domain-general sequence learning as the basis for recursion.
- Constraints on sequence learning influence the cultural evolution of linguistic structures.
- Traditional arguments for a language faculty can be re-evaluated without positing such a faculty.

## Abstract

In the generative tradition, the language faculty has been shrinking—perhaps to include only the mechanism of recursion. This paper argues that even this view of the language faculty is too expansive. We first argue that a language faculty is difficult to reconcile with evolutionary considerations. We then focus on recursion as a detailed case study, arguing that our ability to process recursive structure does not rely on recursion as a property of the grammar, but instead emerges gradually by piggybacking on domain-general sequence learning abilities. Evidence from genetics, comparative work on non-human primates, and cognitive neuroscience suggests that humans have evolved complex sequence learning skills, which were subsequently pressed into service to accommodate language. Constraints on sequence learning therefore have played an important role in shaping the cultural evolution of linguistic structure, including our limited abilities for processing recursive structure. Finally, we re-evaluate some of the key considerations that have often been taken to require the postulation of a language faculty.

## Full-text entities

- **Genes:** DNLZ (DNL-type zinc finger) [NCBI Gene 728489] {aka C9orf151, HEP, HEP1, TIMM15, ZIM17, bA413M3.2}, Foxp2 (forkhead box P2) [NCBI Gene 114142] {aka 2810043D05Rik, CAG-16, D0Kist7}, FOXP2 (forkhead box P2) [NCBI Gene 93986] {aka CAGH44, SPCH1, TNRC10}
- **Diseases:** SLI (MESH:D000080888), and orofacial motor impairments (MESH:D020820), deficit in sequence learning (MESH:D007859), language impairment (MESH:D007806), cognitive impairments (MESH:D003072), Broca (MESH:D001039)
- **Chemicals:** S (MESH:D013455), N (PP) (MESH:C063701), adp (MESH:D000244), V (NP) (PP) (-)
- **Species:** Gallus gallus (bantam, species) [taxon 9031], Homo sapiens (human, species) [taxon 9606], Mus musculus (house mouse, species) [taxon 10090]

## Figures

2 figures with captions in the complete paper: https://tomesphere.com/paper/PMC4550780/full.md

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Source: https://tomesphere.com/paper/PMC4550780