# Electromagnetic Induction Aberration: The Possible Mechanism of Tic Douloureux

**Authors:** Berkley Rish

PMC · DOI: 10.7759/cureus.255 · 2015-03-10

## TL;DR

This paper proposes that electromagnetic induction might explain the pain in trigeminal neuralgia, linking anatomy, physics, and neurology.

## Contribution

The paper introduces a novel theory combining electromagnetic induction with trigeminal neuralgia's pathophysiology.

## Key findings

- Electromagnetic induction at the trigeminal nerve's dorsal root entry zone may cause tic douloureux symptoms.
- The theory extends the vascular compression concept by incorporating physical laws of induction.
- Similar mechanisms might apply to other clinical conditions with analogous features.

## Abstract

A theory based on the principles of electromagnetic induction aberration is presented as the possible mechanism of classic trigeminal neuralgia, tic douloureux.

The anatomy of the dorsal root entry zone of the trigeminal nerve at the pons in the proximity of the superior cerebellar artery presents a scenario conducive to the phenomenon of electromagnetic induction. When the action potentials traversing the axons in this zone of the compromised myelin come into juxtaposition with the vascular structure, the criteria for electromagnetic induction are satisfied. The laws of physics governing the phenomenon indicate that a new current, an aberration, would be produced. This could be responsible for the clinical symptoms of tic douloureux.

Other clinical situations with similar features could share this mechanism. This proposed theory, a merger of anatomy, neurophysiology, and the physics of electromagnetic induction, extends the established concept of vascular compression as the etiology of tic douloureux.

## Linked entities

- **Diseases:** trigeminal neuralgia (MONDO:0008599)

## Full-text entities

- **Diseases:** lateral medullary syndrome (MESH:D014854), cranial nerve syndromes (MESH:D003389), tic pain (MESH:D020323), multiple sclerosis (MESH:D009103), bony lesions (MESH:D000070896), sensory deficits (MESH:D012678), Secondary trigeminal neuralgia (MESH:D014277), cysts (MESH:D003560), vascular compression (MESH:D009408), post-traumatic epilepsy (MESH:D004834), hemorrhage (MESH:D006470), hemifacial spasm (MESH:D019569), neuropathy (MESH:D009422), nociceptive (MESH:D059226), EMIA (MESH:D002869), demyelinating plaques (MESH:D003711), peripheral nerve injuries (MESH:D059348), dysesthesia (MESH:D010292), benign tumors (MESH:D009369), epidermoid cysts (MESH:D004814), AVM (MESH:D001165), hypesthesia (MESH:D006987), pain (MESH:D010146), seizure (MESH:D012640)
- **Species:** Homo sapiens (human, species) [taxon 9606]

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Source: https://tomesphere.com/paper/PMC4494566