# Obliteration of Intercondylar Notch Mimicking Flexion-Extension Gap Imbalance in a Cruciate Retaining Total Knee Arthroplasty

**Authors:** Harun Resit Gungor, Esat Kiter, Semih Akkaya, Nusret Ok, Cagdas Yorukoglu

PMC · DOI: 10.1155/2015/716148 · 2015-06-22

## TL;DR

A patient had ongoing knee pain and limited motion after a knee replacement surgery due to cement blocking the intercondylar notch, which was resolved by removing the cement.

## Contribution

This case highlights an unusual cause of postoperative knee dysfunction related to cement extrusion in the intercondylar notch.

## Key findings

- Extruded cement in the intercondylar notch mechanically blocked terminal extension and limited flexion.
- The issue was resolved by removing the extruded cement and performing polyethylene exchange and patellar resurfacing.

## Abstract

Following total knee arthroplasty (TKA), the most frequent cause of extension deficit and limitation of range of motion in early postoperative period is related to improper tensioning of soft tissues and failure to balance extension and flexion gaps. If a cruciate retaining (CR) prosthesis is the planned implant, then attention should be given to balancing the posterior cruciate ligament (PCL), and any factor that alters this balance may also cause deterioration of knee balance in postoperative period. Here, we report on an unusual case referred from another hospital because of continuous pain and restriction of knee motion in early postoperative period following CR-designed TKA that was initially thought to be due to flexion-extension imbalance. However, during the revision procedure, extruded cement to the intercondylar notch was found to be both mechanically blocking terminal extension and limiting flexion by possible mechanism of irritation of the synovial nerve endings around the stretched anterior fibers of PCL during flexion. This case was successfully treated by removal of extruded cement from intercondylar notch to decompress PCL, polyethylene exchange, and secondary patellar resurfacing.

## Full-text entities

- **Diseases:** lipomas (MESH:D008067), -extension (MESH:D000079822), PCL (MESH:D000070598), extension deficit (MESH:D009461), gap (MESH:C562538), proliferations (MESH:C565054), impingement (MESH:D019534), ganglion cysts (MESH:D045888), intercondylar notch (MESH:D000092483), infection (MESH:D007239), Restriction of knee motion (MESH:D002313), Osteoarthritis (MESH:D010003), Reflex sympathetic dystrophy (MESH:D012019), axial deformity (MESH:C537791), limitation of flexion (MESH:D045745), synovial cysts (MESH:D013581), swelling (MESH:D004487), rheumatoid arthritis (MESH:D001172), effusion (MESH:D000080324), flexion instability (MESH:D043171), pain (MESH:D010146), limitation of motion (MESH:D009041), contractures (MESH:D003286), malalignment (MESH:D017760), TKA (MESH:D007718), Her (MESH:D006013)
- **Species:** Homo sapiens (human, species) [taxon 9606]

## Figures

3 figures with captions in the complete paper: https://tomesphere.com/paper/PMC4491548/full.md

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Source: https://tomesphere.com/paper/PMC4491548