# Correction: Tong et al. Reduced Apoptotic Injury by Phenothiazine in Ischemic Stroke Through the NOX-Akt/PKC Pathway. Brain Sci. 2019, 9, 378

**Authors:** Yanna Tong, Kenneth B. Elkin, Changya Peng, Jiamei Shen, Fengwu Li, Longfei Guan, Yu Ji, Wenjing Wei, Xiaokun Geng, Yuchuan Ding

PMC · DOI: 10.3390/brainsci16030331 · Brain Sciences · 2026-03-20

## Full-text entities

- **Genes:** AKT1 (AKT serine/threonine kinase 1) [NCBI Gene 207] {aka AKT, PKB, PKB-ALPHA, PRKBA, RAC, RAC-ALPHA}, PRRT2 (proline rich transmembrane protein 2) [NCBI Gene 112476] {aka BFIC2, BFIS2, DSPB3, DYT10, EKD1, FICCA}
- **Diseases:** Infarct volume (MESH:D007238), injury to (MESH:D014947), Ischemic Stroke (MESH:D002544), ischemic (MESH:D002545)
- **Chemicals:** promethazine (MESH:D011398), C + P (-), DMSO (MESH:D004121), 2,3,5-Triphenyltetrazolium chloride (MESH:C009591), Phenothiazine (MESH:C031637), chlorpromazine (MESH:D002746)
- **Species:** Homo sapiens (human, species) [taxon 9606], Rattus norvegicus (brown rat, species) [taxon 10116]

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## References

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Source: https://tomesphere.com/paper/PMC13024086