# Correction: Blockade of mTOR ameliorates IgA nephropathy by correcting CD89 and CD71 dysfunctions in humanized mice

**Authors:** Alexandra Cambier, Jennifer Da Silva, Julie Bex, Fanny Canesi, Lison Lachize Neanne, Aurélie Sannier, Hélène Mathieu, Erwan Boedec, Amandine Badie, Renato C. Monteiro

PMC · DOI: 10.1371/journal.pone.0345118 · PLOS One · 2026-03-17

## Full-text entities

- **Genes:** Tfrc (transferrin receptor) [NCBI Gene 22042] {aka 2610028K12Rik, CD71, E430033M20Rik, Mtvr1, TFR, TFR1}, Mtor (mechanistic target of rapamycin kinase) [NCBI Gene 56717] {aka 2610315D21Rik, FRAP, FRAP2, Frap1, RAFT1, RAPT1}
- **Diseases:** cancer (MESH:D009369), IgA nephropathy (MESH:D005922)
- **Species:** Mus musculus (house mouse, species) [taxon 10090]

## Full text

_Full body text omitted from this summary view._ Fetch the complete paper as Markdown: https://tomesphere.com/paper/PMC12994776/full.md

## References

1 references — full list in the complete paper: https://tomesphere.com/paper/PMC12994776/full.md

---
Source: https://tomesphere.com/paper/PMC12994776