# Wealth, health, and happiness: An inverse story of the Easterlin Paradox in China

**Authors:** Yangjie Wang, Lianhua Li, Juan Huang, Hongjie Qiang, Shihe Fu, Chih-Wei Tseng, Chih-Wei Tseng, Chih-Wei Tseng, Chih-Wei Tseng, Chih-Wei Tseng

PMC · DOI: 10.1371/journal.pone.0342445 · 2026-03-12

## TL;DR

A Chinese environmental policy reduced pollution and increased happiness, even though it lowered local incomes, supporting the idea that clean air boosts well-being more than income.

## Contribution

This study provides empirical evidence supporting the environmental explanation of the Easterlin paradox using China's TCZ policy.

## Key findings

- The TCZ policy reduced air pollution in targeted areas.
- The policy increased subjective well-being despite lowering local income.
- The happiness gain from pollution reduction was valued at ￥59.04 per month.

## Abstract

One popular explanation for the Easterlin paradox is that income growth over time is usually accompanied by industrialization and pollution, which cause damage to happiness that cannot be reflected by income change. We examine this explanation by exploring the effects of a large-scale environmental regulation program -the “Two Control Zones (TCZ)” Policy- on subjective well-being (SWB) using data from a series of household surveys in China. We find that, the regulation has successfully mitigated air pollution in the implemented area, although at the cost of local income. Overall, the environmental effect dominates the income effect and TCZ policy increases the SWB of affected people. In particular, despite its negative effect on income, by controlling air pollution, the TCZ policy brought a net increase in residential happiness with a money value of ￥59.04 per month in terms of 2009 CNY. This finding supports the environmental explanation of the Easterlin paradox.

## Full-text entities

- **Genes:** GPHB5 (glycoprotein hormone subunit beta 5) [NCBI Gene 122876] {aka B5, GPB5, ZLUT1}, MS4A1 (membrane spanning 4-domains A1) [NCBI Gene 931] {aka B1, Bp35, CD20, CVID5, FMC7, LEU-16}, H3P16 (H3 histone pseudogene 16) [NCBI Gene 644914] {aka H3.6, H3F3AP6, p21}, C1QBP (complement C1q binding protein) [NCBI Gene 708] {aka COXPD33, GC1QBP, HABP1, SF2AP32, SF2p32, gC1Q-R}, IGKV5-2 (immunoglobulin kappa variable 5-2) [NCBI Gene 28907] {aka B2, IGKV52}
- **Diseases:** respiratory diseases (MESH:D012140), GENERAL (MESH:D004829), CHIP (MESH:C536977), air (MESH:D004618), asthma (MESH:D001249), inflammatory (MESH:D007249), chronic obstructive pulmonary disease (MESH:D029424), bronchiectasis (MESH:D001987), irritation of the conjunctiva and upper respiratory tract (MESH:D012141), MAJOR COMMENTS (MESH:D004830), CHNS (MESH:D044342), lung cancer (MESH:D008175), tuberculosis (MESH:D014376)
- **Chemicals:** sulfur (MESH:D013455), P (MESH:D010758), DeltaWTP^hat (-), SO2 (MESH:D013458)
- **Species:** Homo sapiens (human, species) [taxon 9606]

## Figures

50 figures with captions in the complete paper: https://tomesphere.com/paper/PMC12981521/full.md

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Source: https://tomesphere.com/paper/PMC12981521