# Bilateral Electric Cataracts With Markedly Disparate Onset Times

**Authors:** Loubna Moulahid, Hatim Bazhar, Nabil Bouslous, M.Omar Moustaine

PMC · DOI: 10.7759/cureus.103030 · Cureus · 2026-02-05

## TL;DR

A man developed cataracts in both eyes years apart after high-voltage electric shocks, highlighting the long-term risks of such injuries.

## Contribution

This case report highlights the delayed and bilateral nature of electric cataract development following high-voltage electrocution.

## Key findings

- A 21-year-old male developed a cataract in his right eye years after a high-voltage shock at age seven.
- Phacoemulsification surgery successfully restored vision in the newly affected eye.
- Ocular complications from electrocution can manifest years later and require long-term monitoring.

## Abstract

Ocular injuries secondary to high-voltage electrocution are uncommon and may lead to delayed manifestations, particularly cataract formation. In this article, we describe the case of a 21-year-old male diagnosed with a unilateral electric cataract. The patient had previously undergone cataract surgery in the left eye following a high-voltage electric shock sustained at the age of seven and presented years later with decreased vision in the right eye. Clinical examination revealed an anterior subcapsular cataract in the right eye, with no other associated lesions, particularly involving the retina or the optic nerve head. Prompt therapeutic intervention with phacoemulsification surgery successfully restored normal visual acuity in the affected eye. This case underscores the critical importance of ongoing vigilance in detecting and managing ocular complications following electrocution, even years after the initial incident.

## Linked entities

- **Diseases:** cataract (MONDO:0005129)

## Full-text entities

- **Diseases:** choroidal rupture (MESH:D012421), decreased vision (MESH:D014786), optic atrophy (MESH:D009896), iritis (MESH:D007500), ocular complications (MESH:D008107), inflammatory (MESH:D007249), ocular trauma (MESH:D014947), papilledema (MESH:D010211), optic nerve or chorioretinal abnormalities (MESH:D000080344), nutritional disturbances (MESH:D009748), burn injuries (MESH:D002056), Electrical injuries (MESH:D004556), metabolic disorders (MESH:D008659), retinal detachment (MESH:D012163), conjunctival hyperemia (MESH:D003229), Ocular injuries (MESH:D005131), uveitis (MESH:D014605), Burn scars (MESH:D002921), corneal opacities (MESH:D003318), chorioretinal lesions (MESH:D002825), chorioretinal necrosis or atrophy (MESH:C566236), ocular lesions (MESH:D015821), miosis (MESH:D015877), cataract (MESH:D002386)
- **Chemicals:** oxybuprocaine (MESH:C005298)
- **Species:** Homo sapiens (human, species) [taxon 9606]

## Full text

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## Figures

3 figures with captions in the complete paper: https://tomesphere.com/paper/PMC12967125/full.md

## References

10 references — full list in the complete paper: https://tomesphere.com/paper/PMC12967125/full.md

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Source: https://tomesphere.com/paper/PMC12967125