# Severe Radiographic Legg-Calvé-Perthes Disease With Favorable Long-Term Functional Outcomes Following Conservative Management: A Case Report

**Authors:** Hannah J Grimmett, Stephen E Martiny

PMC · DOI: 10.7759/cureus.102835 · Cureus · 2026-02-02

## TL;DR

A child with severe Legg-Calvé-Perthes disease avoided surgery and achieved good long-term function through non-surgical care.

## Contribution

Demonstrates that conservative management can yield favorable outcomes in severe LCPD cases.

## Key findings

- The patient achieved high functional capacity despite severe femoral head deformity.
- Non-surgical treatment allowed participation in NCAA Division I athletics.
- The patient remains independent in daily activities as a young adult.

## Abstract

Legg-Calvé-Perthes disease (LCPD) is a pediatric disorder characterized by avascular necrosis of the femoral head, with prognosis largely determined by the extent of epiphyseal involvement. Severe radiographic classifications are typically associated with poor long-term outcomes and often prompt surgical intervention. We present a case of a male patient diagnosed with unilateral LCPD at age three with extensive femoral head involvement by age six (Catterall Group 4, Salter-Thompson Group B, Herring Group C) who was managed non-operatively over two decades. Treatment consisted of physiotherapy, activity modification, and pain management without surgical intervention. Despite marked femoral head deformity and residual limitations in hip range of motion, the patient achieved high functional capacity, including participation in National Collegiate Athletic Association (NCAA) Division I athletics, and he maintains independence in activities of daily living as a young adult. This case highlights the potential for favorable functional outcomes with conservative management in carefully selected patients with poor-prognosis LCPD and underscores the importance of individualized treatment planning.

## Linked entities

- **Diseases:** Legg-Calvé-Perthes disease (MONDO:0007885), avascular necrosis (MONDO:0018373)

## Full-text entities

- **Genes:** ALB (albumin) [NCBI Gene 213] {aka FDAHT, HSA, PRO0883, PRO0903, PRO1341}, FGB (fibrinogen beta chain) [NCBI Gene 2244] {aka HEL-S-78p}, IL6 (interleukin 6) [NCBI Gene 3569] {aka BSF-2, BSF2, CDF, HGF, HSF, IFN-beta-2}, CRP (C-reactive protein) [NCBI Gene 1401] {aka PTX1}, HAMP (hepcidin antimicrobial peptide) [NCBI Gene 57817] {aka HEPC, HFE2B, LEAP1, PLTR}
- **Diseases:** avulsion (MESH:D000071562), avascular necrosis of the femoral (MESH:D005271), abnormal osteogenesis (MESH:D010013), Asthma (MESH:D001249), leg length discrepancy (MESH:D007870), LCPD (MESH:D007873), bone mineral loss (MESH:D012080), avascular necrosis (MESH:D010020), ADHD (MESH:D001289), fractures (MESH:D050723), Pain (MESH:D010146), derangement of joint mobility (MESH:D014086), injuries (MESH:D014947), dyslexia (MESH:D004410), inflammation (MESH:D007249), loss (MESH:D016388), tissue injuries (MESH:D017695), valgus (MESH:D060906), ischemia (MESH:D007511), dyscalculia (MESH:D060705), osteoarthritis (MESH:D010003), mushroom deformity (MESH:D009145), thrombophilia (MESH:D019851), sequestration (MESH:D001998), hip and knee pain (MESH:D046788), sclerosis (MESH:D012598), femoral head deformity (MESH:D000070603)
- **Chemicals:** oxygen (MESH:D010100), nonsteroidal anti-inflammatories (-)
- **Species:** Homo sapiens (human, species) [taxon 9606]

## Full text

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## Figures

6 figures with captions in the complete paper: https://tomesphere.com/paper/PMC12959386/full.md

## References

19 references — full list in the complete paper: https://tomesphere.com/paper/PMC12959386/full.md

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Source: https://tomesphere.com/paper/PMC12959386