# Deep-Infiltrating Endometriosis Causing Acute Mechanical Intestinal Obstruction Without Intestinal Invasion: A Case Report with Diagnostic and Surgical Insights

**Authors:** Jung Hyun Park, Jeonghyeon Shin, Mee-Ran Kim

PMC · DOI: 10.3390/jcm15041664 · 2026-02-23

## TL;DR

A rare case of deep-infiltrating endometriosis caused intestinal blockage without invading the bowel, highlighting the need for tailored treatment.

## Contribution

This case expands the known complications of endometriosis by showing obstruction via adhesions, not direct invasion.

## Key findings

- Endometriosis caused acute small bowel obstruction through adhesions, not transmural invasion.
- Laparoscopic surgery and hormonal therapy reduced recurrence risk in this patient.
- Preoperative imaging enabled a minimally invasive approach for diagnosis and treatment.

## Abstract

Background: Endometriosis is a chronic, estrogen-dependent disorder that may extend beyond the pelvis to involve the gastrointestinal tract, most commonly the rectosigmoid and, less frequently, the small bowel. Although often asymptomatic, such lesions may rarely manifest as acute bowel obstruction. Case: We report a 42-year-old woman who presented with small bowel ileus caused by deep-infiltrating endometriosis (DIE). Imaging revealed a right ovarian endometrioma with severe adhesions resulting in a distal ileal transition point. After partial decompression with conservative treatment, laparoscopic adhesiolysis with right salpingo-oophorectomy and left salpingectomy was undertaken. Intraoperative findings revealed dense adnexal–ileal adhesions without transmural involvement. Postoperative hormonal suppression was instituted. Conclusions: This rare case demonstrates small bowel obstruction caused by DIE adhesions without intestinal invasion. Preoperative imaging facilitated a minimally invasive approach, while combined surgical and hormonal therapy was associated with reduced recurrence risk. These findings expand the recognized spectrum of endometriosis-related intestinal complications and support tailored management strategies.

## Linked entities

- **Diseases:** endometriosis (MONDO:0005133)

## Full-text entities

- **Genes:** CRP (C-reactive protein) [NCBI Gene 1401] {aka PTX1}, MUC16 (mucin 16, cell surface associated) [NCBI Gene 94025] {aka CA125}
- **Diseases:** ovarian disease (MESH:D010049), endometrial cyst (MESH:D014591), estrogen-dependent (MESH:D056828), abdominal distension (MESH:D000007), Intestinal obstruction (MESH:D007415), perforations (MESH:D057112), tenderness (MESH:D063806), bowel ileus (MESH:D045823), dyspareunia (MESH:D004414), menorrhagia (MESH:D008595), ascites (MESH:D001201), appendicitis (MESH:D001064), cystic lesion (MESH:D052177), adhesions (MESH:D000267), pelvic pain symptoms (MESH:D017699), dysmenorrhea (MESH:D004412), stricture (MESH:D003251), fever (MESH:D005334), vomiting (MESH:D014839), Intestinal perforation (MESH:D007416), endometriotic lesions (MESH:D009059), malignancy (MESH:D009369), ureteral injury (MESH:D014515), abdominal pain (MESH:D015746), small bowel obstruction (MESH:D007409), acute bowel obstruction (MESH:D010195), injury to (MESH:D014947), ovarian cyst (MESH:D010048), DIE (MESH:D004715)
- **Chemicals:** indigo carmine (MESH:D007203), progesterone (MESH:D011374), Dienogest (MESH:C023635)
- **Species:** Homo sapiens (human, species) [taxon 9606]

## Figures

3 figures with captions in the complete paper: https://tomesphere.com/paper/PMC12942114/full.md

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Source: https://tomesphere.com/paper/PMC12942114