# Successful management of left ventricular high lateral wall rupture during bentall procedure: a case report

**Authors:** Ke Xu, Lailong Sun, Yi Li, Songlin Zhang

PMC · DOI: 10.3389/fcvm.2026.1759056 · Frontiers in Cardiovascular Medicine · 2026-02-09

## TL;DR

A 59-year-old woman survived a rare heart rupture during surgery by combining two repair techniques, showing a successful approach to a life-threatening complication.

## Contribution

Demonstrates successful combined intracardiac and extracardiac repair for left ventricular rupture during a Bentall procedure.

## Key findings

- A combined intracardiac and extracardiac repair successfully managed a left ventricular rupture during a Bentall procedure.
- The patient maintained good cardiac function with no cardiac-related mortality over a 4-year follow-up.

## Abstract

Left ventricular rupture is a highly fatal complication, with reported in-hospital mortality rates as high as 80%, largely due to the challenge of achieving timely surgical intervention. We present the case of a 59-year-old female who underwent a Bentall procedure for moderate-to-severe aortic regurgitation with concomitant aortic sinus and ascending aortic dilation. Shortly after weaning from cardiopulmonary bypass, a sudden rupture of the high lateral left ventricular wall occurred. Successful management involved a combined intracardiac and extracardiac repair: the external tear was first exposed, followed by exploration to identify and suture the corresponding internal defect. Over a 4-year postoperative follow-up, the patient maintained good cardiac function with no cardiac-related mortality. This case highlights the critical therapeutic significance of combined intracardiac and extracardiac repair for left ventricular rupture and underscores key technical lessons for managing this catastrophic event.

## Full-text entities

- **Diseases:** ischemia (MESH:D007511), Atrioventricular Premature Contractions (MESH:D018880), coronary artery stenosis (MESH:D023921), Chamber abnormality (MESH:C535679), Left ventricular enlargement (MESH:D018487), ventricular dilatation (MESH:C566255), aortic valve replacement (MESH:D001024), bleeding (MESH:D006470), Cardiac rupture (MESH:D006341), fatigue (MESH:D005221), chest tightness (MESH:D002637), Marfan syndrome (MESH:D008382), aneurysm (MESH:D000783), motion abnormality (MESH:D009041), shortness of breath (MESH:D004417), cardiogenic shock (MESH:D012770), pseudoaneurysm (MESH:D017541), Left ventricular rupture (MESH:D012421), type A aortic dissection (MESH:D000784), Valvular heart disease (MESH:D006349), myocardial weakness (MESH:D018908), ischemic (MESH:D002545), myocardial disruptions (MESH:D019958), trauma (MESH:D014947), myocardial fibrosis (MESH:D005355), Annulo-aortic ectasia (MESH:D004108), intracardiac defect (MESH:C538262), coronary injury (MESH:D003323), infarction (MESH:D007238), aortic regurgitation (MESH:D001022), dilation of the left ventricle (MESH:D020257), Aortic sinus and ascending aorta dilatation (MESH:D000094630), intra- or postoperative hemorrhage (MESH:D019106), wall (MESH:D056988), Cardiac adhesions (MESH:D006331), aortic root pathologies (MESH:D000094628), Coronary artery injury (MESH:D003324), Aortic dilatation (MESH:D002311), myocardial infarction (MESH:D009203), myocardial ischemia (MESH:D017202), aortic sinus and ascending aortic dilation (MESH:D000094625), dizziness (MESH:D004244), hypertension (MESH:D006973), left ventricular hypertrophy (MESH:D017379)
- **Chemicals:** PTFE (MESH:D011138), heparin (MESH:D006493)
- **Species:** Homo sapiens (human, species) [taxon 9606]

## Full text

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## Figures

4 figures with captions in the complete paper: https://tomesphere.com/paper/PMC12926444/full.md

## References

16 references — full list in the complete paper: https://tomesphere.com/paper/PMC12926444/full.md

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Source: https://tomesphere.com/paper/PMC12926444