Correction: Base-excision repair increases DNA double-strand break clustering within heavy-ion tracks and modulates repair at δ-electron-induced breaks
Laura Schwan, Nicole B. Averbeck, Marco Durante, Burkhard Jakob
Abstract
Genes, proteins, chemicals, diseases, species, mutations and cell lines named across the full text — each resolved to its canonical identifier and authoritative record.
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Taxonomy
TopicsDNA Repair Mechanisms · Radiation Therapy and Dosimetry · Bacterial Genetics and Biotechnology
Correction to: Scientific Reports 10.1038/s41598-025-32823-z, published online 09 January 2026
The original version of this Article contained errors.
In the Materials and methods section, subsection Cell culture and inhibitor treatment the sentence:
“To block the BER pathway, a combination of two inhibitors was used in the cells-culture medium: 10 µM TH5487, an OGG1 inhibitor that prevents OGG1’s binding and repair of 8-oxoG^58,59^ and 40 mM methoxyamine (MX), an indirect APE1 inhibitor, as it makes apurinic and apyrimdinic sites insensitive to APE1^60^.”
now reads:
“To block the BER pathway, a combination of two inhibitors was used in the cells-culture medium: 10 µM TH5487, an OGG1 inhibitor that prevents OGG1’s binding and repair of 8-oxoG^58,59^ and 20 mM methoxyamine (MX), an indirect APE1 inhibitor, as it makes apurinic and apyrimidinic sites insensitive to APE1^60^.”
The original Article has been corrected.