Expression of Concern: Targeting NLRP3 and AIM2 signaling pathways by Viscosol alleviates metabolic dysregulations induced inflammatory responses in diabetic neuro- and nephropathy: An in silico and in vivo study
Abstract
Genes, proteins, chemicals, diseases, species, mutations and cell lines named across the full text — each resolved to its canonical identifier and authoritative record.
Peer Reviews
No public reviews on file for this paper yet. If you reviewed it on a platform where reviews are public (OpenReview, ICLR, NeurIPS, ICML), you can paste yours below so the community can read it here.
Videos
No videos yet. Explain this paper in a talk, walkthrough, or lecture? Add one.
Taxonomy
TopicsInflammasome and immune disorders · Ion Channels and Receptors · Genomics, phytochemicals, and oxidative stress
After this article [1,2] was published, concerns were raised that the article cited as Reference 9 was retracted before publication of [1,2]. Additionally, the PLOS One Editors have concerns about the article’s compliance with the PLOS Authorship policy.
In light of the cumulative issues, the PLOS One Editors issue this Expression of Concern. Readers are advised to interpret the article [1,2] with caution.
The reference list from the paper itself. Each links out to its DOI / PubMed record.
- 1Thahiem S, Ihsan M, Muneer H, Sohail A, Khan M, Murtaza I, et al. Targeting NLRP 3 and AIM 2 signaling pathways by Viscosol alleviates metabolic dysregulations induced inflammatory responses in diabetic neuro- and nephropathy: An in silico and in vivo study. P Lo S One. 2025;20(4):e 0313816. doi: 10.1371/journal.pone.0313816 40173145 PMC 11964203 · doi ↗ · pubmed ↗
- 2Thahiem S, Ihsan M, Muneer H, Sohail A, Khan M, Murtaza I, et al. Correction: Targeting NLRP 3 and AIM 2 signaling pathways by Viscosol alleviates metabolic dysregulations induced inflammatory responses in diabetic neuro- and nephropathy: An in silico and in vivo study. P Lo S One. 2025;20(12):e 0338855. doi: 10.1371/journal.pone.0338855 41379829 PMC 12697987 · doi ↗ · pubmed ↗