# Correction: Bax and Bak can localize to the endoplasmic reticulum to initiate apoptosis

**Authors:** Wei-Xing Zong, Chi Li, Georgia Hatzivassiliou, Tullia Lindsten, Qian-Chun Yu, Junying Yuan, Craig B. Thompson

PMC · DOI: 10.1083/jcb.20030208401202026c · The Journal of Cell Biology · 2026-02-05

## Full-text entities

- **Genes:** BAX (BCL2 associated X, apoptosis regulator) [NCBI Gene 581] {aka BCL2L4}, BAK1 (BCL2 antagonist/killer 1) [NCBI Gene 578] {aka BAK, BAK-LIKE, BCL2L7, CDN1}, COX4I1 (cytochrome c oxidase subunit 4I1) [NCBI Gene 1327] {aka COX IV-1, COX4, COX4-1, COXIV, COXIV-1, MC4DN16}, CANX (calnexin) [NCBI Gene 821] {aka CNX, IP90, P90}
- **Chemicals:** FITC (MESH:D016650), paraformaldehyde (MESH:C003043), thapsigargin (MESH:D019284), PBS (MESH:D007854), brefeldin A (MESH:D020126), tunicamycin (MESH:D014415)
- **Species:** Mus musculus (house mouse, species) [taxon 10090]
- **Cell lines:** MEFs — Mus musculus (Mouse), Finite cell line (CVCL_9115), HM — Homo sapiens (Human), Bladder carcinoma, Cancer cell line (CVCL_9826), HeLa — Homo sapiens (Human), Human papillomavirus-related endocervical adenocarcinoma, Cancer cell line (CVCL_0030), 293T — Homo sapiens (Human), Transformed cell line (CVCL_0063), MCF7 — Homo sapiens (Human), Invasive breast carcinoma of no special type, Cancer cell line (CVCL_0031)

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Source: https://tomesphere.com/paper/PMC12875309