Correction: Association of attenuated leptin signaling pathways with impaired cardiac function under prolonged high-altitude hypoxia
Jianan Wang, Shiying Liu, Lihong Sun, Zhanping Kong, Jiamin Chai, Jigang Wen, Xuan Tian, Nan Chen, Chengli Xu
Abstract
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Taxonomy
TopicsHigh Altitude and Hypoxia · Regulation of Appetite and Obesity · Adipokines, Inflammation, and Metabolic Diseases
Correction to: Scientific Reports 10.1038/s41598-024-59559-6, published online 3 May 2024
The original version of this Article contained an error in Fig. 5K, in which the figure incorrectly uses the immunohistochemistry image of leptin, which should be Ob-Rb. The original Fig. 5 and accompanying legend appear below.Fig. 5. Hypoxia resulted in decreased leptin, ob-Rb, and HIF1α protein levels in rat myocardial tissues. (A) Plasma leptin. (B) Western blot analysis of HIF1α, leptin, and Ob-Rb in left ventricular myocardial tissues. (C–E) Quantification of HIF1α, leptin, and Ob-Rb protein expression in left ventricular myocardial tissues. (F) Western blot analysis of HIF1α, leptin, and Ob-Rb in right ventricular myocardial tissues. (G–I) Quantification of HIF1α, leptin, and Ob-Rb protein expression in left ventricular myocardial tissues. The protein expression levels were normalized to those of β-Tubulin. (J,K) Immunohistochemistry of leptin and Ob-Rb in left and right ventricular myocardial tissues. n = 6. Data are presented as mean ± SEM. The p values are shown on the top of compared group by two-tailed unpaired t-test (for normally distributed data) or Mann–Whitney U test (for nonnormally distributed data).
The original Article has been corrected.