# Corrigendum to “Cannabidiol suppresses proliferation and induces cell death, autophagy and senescence in human cholangiocarcinoma cells via the PI3K/AKT/mTOR pathway” [J Tradition Complement Med 14(6) (2024) 622–634]

**Authors:** Thatsanapong Pongking, Kitti Intuyod, Phonpilas Thongpon, Raynoo Thanan, Chutima Sitthirach, Apisit Chaidee, Suppakrit Kongsintaweesuk, Sirinapha Klungsaeng, Nuttanan Hongsrichan, Chadamas Sakonsinsiri, Kulthida Vaeteewoottacharn, Somdej Kanokmedhakul, Somchai Pinlaor, Porntip Pinlaor

PMC · DOI: 10.1016/j.jtcme.2025.01.001 · Journal of Traditional and Complementary Medicine · 2025-01-02

## Full-text entities

- **Genes:** PIK3CB (phosphatidylinositol-4,5-bisphosphate 3-kinase catalytic subunit beta) [NCBI Gene 5291] {aka P110BETA, PI3K, PI3KBETA, PIK3C1}, MTOR (mechanistic target of rapamycin kinase) [NCBI Gene 2475] {aka FRAP, FRAP1, FRAP2, RAFT1, RAPT1, SKS}, AKT1 (AKT serine/threonine kinase 1) [NCBI Gene 207] {aka AKT, PKB, PKB-ALPHA, PRKBA, RAC, RAC-ALPHA}
- **Diseases:** cholangiocarcinoma (MESH:D018281)
- **Chemicals:** Cannabidiol (MESH:D002185)
- **Species:** Homo sapiens (human, species) [taxon 9606]

## Full text

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Source: https://tomesphere.com/paper/PMC12624692