# Effects of Apnoea versus Normal Breathing on Physiological Responses during High-Intensity Interval Training in Swimming

**Authors:** Pinelopi Liapaki, Helen Soultanakis, Ioannis Kalomenidis, Vicente Javier Clemente-Suárez, Stamatis Mourtakos, Spyros Zakynthinos

PMC · DOI: 10.5114/jhk/195587 · Journal of Human Kinetics · 2025-05-26

## TL;DR

This study compares the effects of normal breathing versus holding breath during high-intensity swimming, finding that holding breath improves performance despite increased acidosis.

## Contribution

The study reveals that apnoea improves sprint performance in swimming without compensating for metabolic acidosis.

## Key findings

- Apnoea leads to faster 50-meter freestyle swimming times compared to normal breathing.
- Uncompensated metabolic acidosis is more pronounced during apnoea.
- Heart rate decreases during apnoea, but performance improves.

## Abstract

The objective of this research was to examine the impact of conventional breathing versus apnoea technique on acid-base equilibrium, physiological reactions, and performance throughout high-intensity interval training sessions in swimming. Two groups of sixteen athletes completed 6 x 50-m intervals of freestyle swimming with normal breathing and apnoea at maximum intensity, with a 1-min rest interval. Capillary blood gasses (pH, PCO2, PO2, HCO3, Hct, Hb) were collected at four measurement time points: 1) at rest, 2) at rest just after the 3rd repetition, 3) at finish, and 4) at the 10th min of recovery. Measured variables included the heart rate (HR) during swimming, lactate (La) concentration and swimming time (t50). Uncompensated metabolic acidosis, exhibiting greater prominence during apnoea, was attributed to heightened lactic acidosis under both breathing conditions. Despite experiencing bradycardia, swimmers demonstrated faster completion times during apnoea. In conclusion, during repeated high-intensity short-distance swimming, specifically 50 m of freestyle, apnoea enhances sprint performance without compensating for metabolic acidosis.

## Full-text entities

- **Diseases:** lactic acidosis (MESH:D000140), bradycardia (MESH:D001919), metabolic acidosis (MESH:D000138), Apnoea (MESH:D001049)
- **Chemicals:** La (MESH:D019344), Hct (MESH:D006852), HCO3 (MESH:D001639), PO2 (MESH:C093415), PCO2 (-)

## Full text

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## Figures

4 figures with captions in the complete paper: https://tomesphere.com/paper/PMC12612829/full.md

## References

41 references — full list in the complete paper: https://tomesphere.com/paper/PMC12612829/full.md

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Source: https://tomesphere.com/paper/PMC12612829