# Unraveling the Link Between Trauma and Problematic Alcohol Use Over Time: The Influence of Executive Function and Social Cognition Deficits

**Authors:** Yi‐Ying Lu, Ming‐Hong Hsieh, Triantoro Safaria, Yu‐Lien Huang

PMC · DOI: 10.1002/cpp.70169 · Clinical Psychology & Psychotherapy · 2025-11-06

## TL;DR

This study shows that trauma-related stress can lead to alcohol misuse by impairing cognitive and social skills, suggesting that improving these skills may help reduce alcohol use.

## Contribution

The study identifies executive function and social cognition as mediators between trauma and alcohol use, offering new insights into intervention strategies.

## Key findings

- PTSS at Time 1 directly predicted PAU at both Time 1 and Time 2.
- PTSS indirectly influenced PAU through deficits in EF, emotion recognition, and ToM.
- Cognitive and social-cognitive impairments are key risk factors for alcohol misuse among trauma-exposed individuals.

## Abstract

The co‐occurrence of posttraumatic stress symptoms (PTSS) and problematic alcohol use (PAU) is well established, yet the mechanisms linking them remain unclear. Trauma may impair executive function (EF) and social cognition, such as emotion recognition and theory of mind (ToM), which hinder emotional regulation and increase reliance on alcohol as a coping strategy. This study examined whether deficits in EF and social cognition mediate the relationship between PTSS and PAU. A two‐wave longitudinal design was employed with 200 Taiwanese adults exposed to trauma recruited from the community and psychiatric clinics. At baseline (Time 1), participants completed self‐report measures of trauma, PTSS and PAU, along with computerized tasks assessing EF, emotion recognition and ToM. One month later (Time 2), PTSS and PAU were reassessed in 143 participants. PTSS at Time 1 directly predicted PAU at both Time 1 and Time 2, supporting the self‐medication hypothesis. In addition, PTSS indirectly influenced PAU through deficits in EF, emotion recognition and ToM. These findings suggest that cognitive and social‐cognitive impairments are key risk factors for alcohol misuse among trauma‐exposed individuals. Interventions targeting EF and social cognition may therefore help reduce PAU and support trauma recovery.

Posttraumatic stress symptoms are associated with problematic alcohol use, supporting the self‐medication hypothesis.Trauma can impair executive function and social cognition, including emotion recognition and theory of mind.These cognitive and emotional deficits may increase vulnerability to alcohol misuse among trauma‐exposed individuals.Interventions targeting executive function and social cognition could help reduce problematic alcohol use in this population.

Posttraumatic stress symptoms are associated with problematic alcohol use, supporting the self‐medication hypothesis.

Trauma can impair executive function and social cognition, including emotion recognition and theory of mind.

These cognitive and emotional deficits may increase vulnerability to alcohol misuse among trauma‐exposed individuals.

Interventions targeting executive function and social cognition could help reduce problematic alcohol use in this population.

## Full-text entities

- **Diseases:** Trauma (MESH:D014947), Social Cognition Deficits (MESH:D003072), PTSS (MESH:D013313), PAU (MESH:D000437), emotion recognition (MESH:D020238), psychiatric (MESH:D001523), deficits in EF (MESH:D001289)
- **Chemicals:** alcohol (MESH:D000438)

## Full text

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## Figures

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## References

60 references — full list in the complete paper: https://tomesphere.com/paper/PMC12591354/full.md

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Source: https://tomesphere.com/paper/PMC12591354