# Recurring Paralysis and a Race Against Time: A Case of Cauda Equina Syndrome With Delayed Diagnosis and Incomplete Neurological Recovery

## TL;DR

A case study describes a man with cauda equina syndrome who experienced delayed diagnosis and incomplete recovery, emphasizing the need for early detection and careful monitoring.

## Contribution

This paper contributes a detailed clinical case highlighting the challenges of diagnosing and managing cauda equina syndrome with atypical and recurrent symptoms.

## Key findings

- Delayed diagnosis of CES led to incomplete neurological recovery despite surgical intervention.
- The patient required a second surgery due to recurrent and contralateral symptoms.
- Early recognition and postoperative vigilance are critical for preventing irreversible disability in CES.

## Abstract

Cauda equina syndrome (CES) is a rare but critical neurological condition caused by compression of the cauda equina, a collection of lumbosacral nerve roots responsible for lower limb function along with bowel and bladder control. While CES often presents as a sudden and severe onset of symptoms, it can also develop insidiously, mimicking other conditions in its early stages, such as lumbar radiculopathy, spinal stenosis, or urinary tract disorders, often contributing to delayed recognition. Given the variability in clinical presentation, early recognition of suggestive signs and symptoms is crucial to prevent permanent neurological deficits. Delayed recognition may result in irreversible disability despite surgical intervention. We report the case of a 35-year-old male patient with a history of chronic right-sided sciatica, who reported to the emergency department with progressively worsening lower back pain radiating to the right leg, accompanied by bilateral limb weakness and evolving neurological deficits. Urgent MRI imaging revealed a significant L4-L5 disc herniation compressing the cauda equina, prompting emergency surgical decompression. Although the patient initially improved following spinal decompression, he later re-presented with recurrent and contralateral symptoms, ultimately requiring a second surgical intervention (lumbar microdiscectomy). This case highlights the importance of early recognition of CES symptoms and the need for vigilant postoperative monitoring to detect recurrent or contralateral symptoms. Atypical presentations of CES should also be acknowledged to avoid delayed or missed diagnosis of this potentially devastating neurological condition.