Author Correction: Bmi-1 promotes the proliferation, migration and invasion, and inhibits cell apoptosis of human retinoblastoma cells via RKIP
Qian Li, Te Fu, Ning Wei, Qiaoling Wang, Xin Zhang
Abstract
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TopicsCancer Mechanisms and Therapy · Melanoma and MAPK Pathways · Signaling Pathways in Disease
Correction to: Scientific Reports 10.1038/s41598-024-65011-6, published online 24 June 2024
The original version of the Article contained an error in Figure 1. As a result of an error during figure assembly, the image for the Scramble condition for SO-RB50 in Figure 1E was mistakenly a duplication of the image for the siBmi-1 + siRKIP for SO-RB50 in Figure 4C.
The original Figure 1 and accompanying legend appear below.
Fig. 1. Knockdown of Bmi-1 inhibits cell proliferation, migration and invasion, and increases cell apoptosis. RT-qPCR (A) and Western blot (B) showed that the expression of Bmi-1 was higher in Y79, SO-RB50 and Weri-RB1 cells than that of normal retinal vascular endothelial cell line ACBRI-181. (C) MTT assay revealed that knockdown of Bmi-1 suppressed cell proliferation compared with control group at 24 h, 48 h and 72 h. (D) Wound healing displayed that Bmi-1 silencing reduced the migration of SO-RB50 cells (Scale bar: 500 μm) and Weri-RB1 cells (Scale bar: 500 μm). (E) Transwell assay displayed that Bmi-1 silencing reduced the invasion of SO-RB50 cells and Weri-RB1 cells (Scale bar: 200 μm). (F) FCM demonstrated that knockdown of Bmi-1 increased cell apoptosis. *P < 0.05, **P < 0.01, ***P < 0.001, compared to the ACBRI-181 cells or the Scramble group.
The original Article has been corrected.