# Physical Activity as a Lifestyle Intervention for Type 1 Diabetics: A Comprehensive Review

**Authors:** Noha A Alzahrani, Ihdaa J Abdulwahab, Moteab S Alotaybi, Abdulaziz Albogami

PMC · DOI: 10.7759/cureus.87351 · Cureus · 2025-07-05

## TL;DR

This review explores how physical activity affects body composition and lifestyle in type 1 diabetes patients.

## Contribution

The study highlights the benefits of resistance and aerobic training on body composition in type 1 diabetics.

## Key findings

- Resistance exercise increases muscle mass and reduces fat mass in T1DM patients.
- Higher intensity and duration of physical activity improve fat-free mass.
- Parental support and community knowledge are crucial for encouraging physical activity in T1DM patients.

## Abstract

The aim of this study was to assess the influence of aerobic and resistance training on anthropometric measures such as body mass index, body weight, fat mass, and fat-free mass among type 1 diabetics. A thorough literature review was performed on PubMed. Our search yielded 196 articles. We included eight studies that dealt with type 1 diabetes mellitus (T1DM) patients who met our inclusion criteria and consisted of randomized controlled trials, systematic reviews, and meta-analyses.

This review article found that resistance exercise increased muscle mass and reduced fat mass in T1DM patients. Increasing the duration and intensity of physical activity led to an increase in fat-free mass. Parental support among type 1 diabetic children showed positive attitudes towards a healthy lifestyle. Addressing stigma among adolescents with T1DM is important, and personal and community knowledge about the condition can help facilitate physical activity participation. Collaboration between healthcare providers, families, and peers is essential in managing physical activity in T1DM patients. Having a staff member with specific training to provide physical activity advice can benefit diabetes teams. Further research over a longer duration is necessary to confirm these outcomes.

## Linked entities

- **Diseases:** type 1 diabetes mellitus (MONDO:0005147), T1DM (MONDO:0005147)

## Full-text entities

- **Genes:** INS (insulin) [NCBI Gene 3630] {aka IDDM, IDDM1, IDDM2, ILPR, IRDN, MODY10}, FAT1 (FAT atypical cadherin 1) [NCBI Gene 2195] {aka CDHF7, CDHR8, FAT, ME5, hFat1}
- **Diseases:** hypoxic (MESH:D002534), T1DM (MESH:D003922), underweight (MESH:D013851), Diabetes (MESH:D003920), muscle (MESH:D019042), type 2 diabetes (MESH:D003924), BMI (MESH:C536030), muscle hypertrophy (MESH:C536106), cardiovascular complications (MESH:D002318), hypoglycemia (MESH:D007003), DM (MESH:D009223), Myopathy (MESH:D009135), PA (MESH:D059445), muscular failure (MESH:D051437)
- **Chemicals:** blood glucose (MESH:D001786), oxygen (MESH:D010100), ATP (MESH:D000255), glucose (MESH:D005947), cholesterol (MESH:D002784), rapid (-), carbohydrate (MESH:D002241), lactate (MESH:D019344)
- **Species:** Homo sapiens (human, species) [taxon 9606]
- **Mutations:** S625X

## Full text

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## References

47 references — full list in the complete paper: https://tomesphere.com/paper/PMC12321317/full.md

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Source: https://tomesphere.com/paper/PMC12321317