Correction to “Defective Dimerization of FoF1‐ATP Synthase Secondary to Glycation Favors Mitochondrial Energy Deficiency in Cardiomyocytes During Aging”

Abstract
Genes, proteins, chemicals, diseases, species, mutations and cell lines named across the full text — each resolved to its canonical identifier and authoritative record.
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Taxonomy
TopicsMitochondrial Function and Pathology · Metabolism and Genetic Disorders · Coenzyme Q10 studies and effects
Bou‐Teen, D., Fernandez‐Sanz, C., Miro‐Casas, E., Nichtova, Z., Bonzon‐Kulichenko, E., Casós, K., Inserte, J., Rodriguez‐Sinovas, A., Benito, B., Sheu, S.‐S., Vázquez, J., Ferreira‐González, I., & Ruiz‐Meana, M. (2022). Defective dimerization of FoF1‐ATP synthase secondary to glycation favors mitochondrial energy deficiency in cardiomyocytes during aging. Aging Cell, 21, e13564. https://doi.org/10.1111/acel.13564
In Funding Information, the text “This work was supported by the Instituto de Salud Carlos III of the Spanish Ministry of Health (FIS‐PI19‐01196) and a grant from the Sociedad Española de Cardiología (SEC/FEC‐INV‐BAS 217003)” was insufficient.
This should have read
This work was funded by the Instituto de Salud Carlos III of the Spanish Ministry of Health (FIS‐PI19/01196) and co‐funded by the European Union (FEDER) and a grant from the Sociedad Española de Cardiología (SEC/FEC‐INV‐BAS 217003).
