# Correction: The absence of Pitx3 results in postnatal loss of dopamine neurons and is associated with an increase in the pro-apoptotic Bcl2 factor Noxa and cleaved caspase 3

**Authors:** Willemieke M. Kouwenhoven, Edward J. Robinson, Daniek Hamberg, Lars von Oerthel, Marten P. Smidt, Lars P. van der Heide

PMC · DOI: 10.1038/s41419-025-07729-3 · Cell Death & Disease · 2025-06-17

## Full-text entities

- **Genes:** BCL2 (BCL2 apoptosis regulator) [NCBI Gene 596] {aka Bcl-2, PPP1R50}, PMAIP1 (phorbol-12-myristate-13-acetate-induced protein 1) [NCBI Gene 5366] {aka APR, NOXA}, CASP3 (caspase 3) [NCBI Gene 836] {aka CPP32, CPP32B, SCA-1}, PITX3 (paired like homeodomain 3) [NCBI Gene 5309] {aka ASGD1, ASMD, ASOD, CTPP4, CTRCT11}
- **Chemicals:** dopamine (MESH:D004298)

## Full text

_Full body text omitted from this summary view._ Fetch the complete paper as Markdown: https://tomesphere.com/paper/PMC12174352/full.md

---
Source: https://tomesphere.com/paper/PMC12174352