# Chip Formation Mechanisms When Cutting Amorphous Alloy with Cubic Boron Nitride Tools Based on Constitutive Equation Parameter Optimisation

**Authors:** Jinguang Du, Dingkun Wang, Yaoxuan Guo, Wuyi Ming, Wenbin He

PMC · DOI: 10.3390/mi16050534 · Micromachines · 2025-04-29

## TL;DR

This paper improves the accuracy of cutting simulations for amorphous alloys by optimizing a constitutive model and analyzing chip formation mechanisms.

## Contribution

A novel parameter optimization approach for the Johnson-Cook model is introduced using Oxley’s cutting theory and particle swarm optimization.

## Key findings

- The optimized model reduced simulation errors for cutting forces to 12.461% and 9.161%.
- Chip transformation from lamellar to serrated was linked to plastic deformation and adiabatic shear.
- Temperature, strain rate, and stress variations were analyzed using the optimized constitutive model.

## Abstract

Owing to potential inaccuracies in the current stress–strain curve used for constructing the Johnson–Cook (JC) constitutive model of amorphous alloys, the parameters of the JC constitutive equation were derived using Oxley’s cutting theory, negative chamfer theoretical mechanics modelling, and the particle swarm optimisation algorithm. A two-dimensional finite element cutting model was subsequently established using AdvantEdge software. The optimised constitutive model was used to simulate the main cutting force (Fz) and the backward force (Fy), which resulted in average errors of 12.461% and 9.161%, respectively. Based on the optimised constitutive model in which the JC constitutive equation parameters were derived using Oxley’s method, the variations in temperature, strain rate, and stress in the deformation zone during the cutting process were analysed. The chip microstructures revealed the transformation of lamellar chips into serrated chips resulting from a combination of plastic deformation, adiabatic shear, and shear slip.

## Full-text entities

- **Genes:** FBXO11 (F-box protein 11) [NCBI Gene 80204] {aka FBX11, IDDFBA, UBR6, UG063H01, VIT1}, AKTIP (AKT interacting protein) [NCBI Gene 64400] {aka FT1, FTS}
- **Diseases:** CO (MESH:D002303), stress fracture (MESH:D015775), injury to (MESH:D014947), FEMs (MESH:D004195)
- **Chemicals:** CBN (-), copper (MESH:D003300), TiN (MESH:D014001)
- **Species:** Homo sapiens (human, species) [taxon 9606]

## Full text

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## Figures

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## References

46 references — full list in the complete paper: https://tomesphere.com/paper/PMC12114555/full.md

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Source: https://tomesphere.com/paper/PMC12114555