Correction: Transitioning from the Safety of the Womb to the Outside World for Neonates with Life-Threatening Cardiovascular Conditions: The IMmediate Postpartum Access to Cardiac Therapy (IMPACT) Procedure
Asif Padiyath, Jennifer M. Lynch, Lisa M. Montenegro, Susan C. Nicolson, Olivia Nelson, Anita L. Szwast, Amanda J. Shillingford, Christine B. Falkensammer, Jill J. Savla, Julie Moldenhauer, Nahla Khalek, Jack Rychik

Abstract
Genes, proteins, chemicals, diseases, species, mutations and cell lines named across the full text — each resolved to its canonical identifier and authoritative record.
Peer Reviews
No public reviews on file for this paper yet. If you reviewed it on a platform where reviews are public (OpenReview, ICLR, NeurIPS, ICML), you can paste yours below so the community can read it here.
Videos
No videos yet. Explain this paper in a talk, walkthrough, or lecture? Add one.
Taxonomy
TopicsCardiovascular Issues in Pregnancy · Congenital Heart Disease Studies · Cardiac Arrest and Resuscitation
Correction to: Pediatric Cardiology 10.1007/s00246-025-03775-y
In Table 1 of this article, the data in the fifth row of the first column was incorrectly stated as “class IIIa” and should have been “class IIIb”. The incorrect and the corrected versions of Table 1 are given below.
Incorrect Table: Table 1. Severity classification for delivery of fetuses with cardiovascular disease at The Children’s Hospital of Philadelphia—Special Delivery UnitDefinitionPGE?ActionAccessExample anomalies/diseases*CLASS IaSimple cardiovascular anomaly or disease, requiring delivery in the SDU but no hemodynamic instability anticipated at deliveryNONeonatology to manage deliveryPIV, no UAC∙ LV/RV size discrepancy with suspicion of possible Coarctation of the aorta∙ AV Canal, balanced∙ TOF with mild PS (“Pink TOF”)∙ Benign arrhythmia (non-sustained SVT, no hydrops, no ventricular dysfunction)CLASS IbNeonatology to manage deliveryUVC + UAC∙ Truncus Arteriosus∙ LV-RV disproportion, with intent to test ductal closure for possible coarctation of aortaCLASS IICardiovascular anomaly or disease of moderate severity, including ductal dependent lesions. Hemodynamic instability is possible, but unlikely and not anticipated at deliveryYESNeonatology to manage deliveryUVC + UAC∙ HLHS, no risk factors (open inter-atrial communication, no significant TR, good RV function)∙ Single ventricle with critical (suspect ductal dependent) pulmonary outflow obstruction (e.g., tricuspid atresia)∙ Single ventricle with critical (suspect ductal dependent) systemic outflow obstruction (e.g., Double-inlet LV with arch obstruction)∙ Coarctation of the aorta∙ Critical aortic or pulmonic stenosis∙ TOF with moderate, or severe PS (ductal dependent)∙ TOF with pulmonary atresia∙ Pulmonary atresia with intact ventricular septumCLASS IIIaCardiovascular anomaly or disease of important severity with the possibility or likelihood of hemodynamic instability at deliveryYESNeonatology to manage delivery in collaboration with CardiologyUVC + UAC∙ Most transposition of the great arteries (see Class IV category for exception)∙ HLHS with moderate atrial level restriction, or significant tricuspid regurgitationCLASS IIIaNONeonatology to manage delivery in collaboration with Cardiology∙ Total anomalous pulmonary venous connection (see Class IV category for exception)∙ Ebstein’s anomaly or tricuspid valve dysplasia with severe tricuspid regurgitation∙ TOF with Absent Pulmonary Valve Leaflet Syndrome∙ CHD with evidence for ventricular dysfunction∙ CHD with evidence for significant AV valve insufficiency∙ Sustained tachyarrhythmia such as SVTIMPACT (CLASS IV)Cardiovascular anomaly or disease in which hemodynamic instability is anticipated once separated from placental circulation thus IMmediate Post-partum Access to Cardiac Therapy** (IMPACT)** is implemented for urgent life-saving careYES or NO, DEPENDS ON ANOMALYCardiac services including cardiac anesthesiology and Fetal Heart program to manage delivery and interventionUVC + UAC∙ HLHS with highly restrictive or intact atrial septum∙ Fetus with complete heart block requiring pacing∙ Hydropic fetus with cardiovascular disease∙ Transposition of the great arteries, intact ventricular septum and evident prenatal restriction of the foramen ovale∙ Total anomalous pulmonary venous connection with concern of obstruction∙ Ebstein’s anomaly or severe tricuspid valve dysplasia, with suspected pulmonary hypoplasia or anticipated respiratory insufficiency∙ TOF with Absent Pulmonary Valve Leaflet Syndrome and anticipated respiratory insufficiency
Corrected Table: Table 1. Severity classification for delivery of fetuses with cardiovascular disease at The Children’s Hospital of Philadelphia—Special Delivery UnitDefinitionPGE?ActionAccessExample anomalies/diseases*CLASS IaSimple cardiovascular anomaly or disease, requiring delivery in the SDU but no hemodynamic instability anticipated at deliveryNONeonatology to manage deliveryPIV, no UAC∙ LV/RV size discrepancy with suspicion of possible Coarctation of the aorta∙ AV Canal, balanced∙ TOF with mild PS (“Pink TOF”)∙ Benign arrhythmia (non-sustained SVT, no hydrops, no ventricular dysfunction)CLASS IbNeonatology to manage deliveryUVC + UAC∙ Truncus Arteriosus∙ LV-RV disproportion, with intent to test ductal closure for possible coarctation of aortaCLASS IICardiovascular anomaly or disease of moderate severity, including ductal dependent lesions. Hemodynamic instability is possible, but unlikely and not anticipated at deliveryYESNeonatology to manage deliveryUVC + UAC∙ HLHS, no risk factors (open inter-atrial communication, no significant TR, good RV function)∙ Single ventricle with critical (suspect ductal dependent) pulmonary outflow obstruction (e.g., tricuspid atresia)∙ Single ventricle with critical (suspect ductal dependent) systemic outflow obstruction (e.g., Double-inlet LV with arch obstruction)∙ Coarctation of the aorta∙ Critical aortic or pulmonic stenosis∙ TOF with moderate, or severe PS (ductal dependent)∙ TOF with pulmonary atresia∙ Pulmonary atresia with intact ventricular septumCLASS IIIaCardiovascular anomaly or disease of important severity with the possibility or likelihood of hemodynamic instability at deliveryYESNeonatology to manage delivery in collaboration with CardiologyUVC + UAC∙ Most transposition of the great arteries (see Class IV category for exception)∙ HLHS with moderate atrial level restriction, or significant tricuspid regurgitationCLASS IIIbNONeonatology to manage delivery in collaboration with Cardiology∙ Total anomalous pulmonary venous connection (see Class IV category for exception)∙ Ebstein’s anomaly or tricuspid valve dysplasia with severe tricuspid regurgitation∙ TOF with Absent Pulmonary Valve Leaflet Syndrome∙ CHD with evidence for ventricular dysfunction∙ CHD with evidence for significant AV valve insufficiency∙ Sustained tachyarrhythmia such as SVTIMPACT (CLASS IV)Cardiovascular anomaly or disease in which hemodynamic instability is anticipated once separated from placental circulation thus IMmediate Post-partum Access to Cardiac Therapy** (IMPACT)** is implemented for urgent life-saving careYES or NO, DEPENDS ON ANOMALYCardiac services including cardiac anesthesiology and Fetal Heart program to manage delivery and interventionUVC + UAC∙ HLHS with highly restrictive or intact atrial septum∙ Fetus with complete heart block requiring pacing∙ Hydropic fetus with cardiovascular disease∙ Transposition of the great arteries, intact ventricular septum and evident prenatal restriction of the foramen ovale∙ Total anomalous pulmonary venous connection with concern of obstruction∙ Ebstein’s anomaly or severe tricuspid valve dysplasia, with suspected pulmonary hypoplasia or anticipated respiratory insufficiency∙ TOF with Absent Pulmonary Valve Leaflet Syndrome and anticipated respiratory insufficiency
The original article has been corrected.
