Corrigendum to “The ACE2/Ang-(1-7)/MasR axis alleviates brain injury after cardiopulmonary resuscitation in rabbits by activating PI3K/Akt signaling”

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Corrigendum to J. Cheng, H. Yang, F. Chen, L. Qiu, F. Chen, Y. Du and X. Meng. The ACE2/Ang-(1-7)/MasR axis alleviates brain injury after cardiopulmonary resuscitation in rabbits by activating PI3K/Akt signaling. Transl Neurosci. 2024;15(1):20220334. https://doi.org/10.1515/tnsci-2022-0334.

After publishing this article, the authors found the errors in the article text, which are corrected as follows:

1. The scoring criteria in Table 1 were wrongly written. The correct version is given below.

2. The caption of Figure 1 was incorrect, which should be changed to: The NDS was significantly decreased following CA/CPR induction, which was improved by Ang-(1-7) administration, whereas A779 administration in the presence of Ang-(1-7) significantly reduced the NDS (a). Then, brain water content was measured, which was increased after CA/CPR; Ang-(1-7) reversed this increase, whereas A779 attenuated the inhibitory effect of Ang-(1-7) (1b). NSE is an intracellular enzyme located within neurons and neuroectodermal cells, and S100B protein is located in astrocytes and Schwann cells [36]. Increases in NSE and S100B indicate neuronal damage [37]. Ang-(1-7) administration abolished the increase in serum NSE and S100B levels at 72 h after resuscitation, whereas A779 treatment increased the serum levels of NSE and S100B in the context of Ang-(1-7) administration (c and d). Overall, MasR activation alleviates CA/CPR-induced neurological deficits, brain edema, and neuronal damage, and its inhibition has the opposite effects.

3. The ordinate of Figure 1d should be changed to S100B.

The authors would like to apologize for any inconvenience caused.