Corrigendum: Glaucoma and microglia-induced neuroinflammation
Makoto Ishikawa, Yukitoshi Izumi, Kota Sato, Taimu Sato, Charles F. Zorumski, Hiroshi Kunikata, Toru Nakazawa
Abstract
Genes, proteins, chemicals, diseases, species, mutations and cell lines named across the full text — each resolved to its canonical identifier and authoritative record.
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Taxonomy
TopicsGlaucoma and retinal disorders · Flavonoids in Medical Research
Incorrect Reference
In the published article, one of the references has been retracted. This article has been removed, and replaced with the following article: Tremblay MÈ, Stevens B, Sierra A, Wake H, Bessis A, Nimmerjahn A. The role of microglia in the healthy brain. J Neurosci (2011) 31(45):16064-9. doi: 10.1523/JNEUROSCI.4158-11.2011
The article text has also been updated to reflect this change. The first paragraph of Section 2, Functions of Microglia in the Retina, previously read as follows:
“Microglia are thought to derive from monocytes that enter the retina from the blood stream during development, and dynamically move their cellular projections even under physiological conditions (12), making physical contact with neurons and synapses and performing synaptic pruning to remove unnecessary synapses. In the development of excitatory circuits, synaptic pruning, by which extrasynaptic connections are eliminated by microglia, is thought to occur when complement C1q is expressed at synapses by TGF-β secreted from astrocytes (13).”
The corrected sentence appears below:
“Microglia are thought to derive from monocytes that enter the retina from the blood stream during development, and dynamically move their cellular projections even under physiological conditions (12), making physical contact with neurons and synapses and performing synaptic pruning to remove unnecessary synapses (13).”
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.