Correction to: Cardiac sarcomere mechanics in health and disease
Claudia Crocini, Michael Gotthardt
Abstract
Genes, proteins, chemicals, diseases, species, mutations and cell lines named across the full text — each resolved to its canonical identifier and authoritative record.
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Taxonomy
TopicsCardiac Structural Anomalies and Repair
Correction to: Biophysical Reviews (2021) 13:637–652
https://doi.org/10.1007/s12551-021-00840-7
In Fig. 2B of this article’s original version, some of the mechanisms for changing titin stiffness were accidentally inverted. The corrected version of Fig. 2B is shown below.Fig. 2. Titin-based passive tension. A Developmental changes in titin isoform expression facilitate the transition from fetal to adult force generation. B The relationship of sarcomere length and passive tension changes in disease via posttranslational modifications (protein kinases PKA, PKC, PKG acting on the titin spring region) and changes in titin isoform expression (N2BA to N2B titin isoform ratio)