Author Correction: Toll‐like receptor 9 protects non‐immune cells from stress by modulating mitochondrial ATP synthesis through the inhibition of SERCA2
Yasunori Shintani, Hannes CA Drexler, Hidetaka Kioka, Cesare MN Terracciano, Steven R Coppen, Hiromi Imamura, Masaharu Akao, Junichi Nakai, Ann P Wheeler, Shuichiro Higo, Hiroyuki Nakayama, Seiji Takashima, Kenta Yashiro, Ken Suzuki
Abstract
Genes, proteins, chemicals, diseases, species, mutations and cell lines named across the full text — each resolved to its canonical identifier and authoritative record.
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Taxonomy
TopicsEicosanoids and Hypertension Pharmacology · interferon and immune responses · Immune Response and Inflammation
Correction to: EMBO Reports (2014) 15:438–445. 10.1002/embr.201337945 | Published online 7 March 2014
The authors contacted the Journal after a panel re-use was identified in the manuscript without proper citation.
Figure 2B is retracted. Associated text is updated.
The following text has been updated.
Page 2 of manuscript PDF.
Finally, immunofluorescence studies demonstrated that not only the overexpressed TLR9-HA-FLAG protein colocalized with SERCA2 (panel 3A—in reference 6) and with the ER/SR marker KDEL in cardiomyocytes (Fig 2B and Supplementary Fig S1C online), but also the endogenous TLR9 colocalized with SERCA2 (Fig 2C). Collectively from these data, we conclude that TLR9 interacts with SERCA2 at ER/SR in cardiomyocytes.
Author Comments:
It has recently been brought to our attention that we reused the panels in Figure 2B from our paper published earlier (Figure 3A, upper, 0 min. PNAS. 2013;110(13):5109-14. 10.1073/pnas.1219243110). We therefore remove the Figure 2B panels and revise the corresponding manuscript text.
This error does not affect the conclusions of the original paper. All authors agree to this correction and apologize for this error.