# Emerging Trends in Pathophysiology: Insights from the 9th International Congress of the International Society for Pathophysiology (ISP 2023)

**Authors:** Anatolii Kubyshkin, Sergey Bolevich, Leonid Churilov, Vladimir Jakovljevic, Evgeniia Kovalenko, Aleksandr Korovin

PMC · DOI: 10.3390/pathophysiology31010011 · Pathophysiology · 2024-03-04

## TL;DR

This paper summarizes key research trends and findings presented at the 9th International Congress of the International Society for Pathophysiology.

## Contribution

The paper highlights new pathogenic pathways and molecular mechanisms identified through recent pathophysiological research.

## Key findings

- New pathogenic pathways were investigated at the ISP 2023 congress.
- Cellular and molecular mechanisms, along with target molecules, were identified.
- New therapeutic strategies were discussed by leading scientific teams.

## Abstract

This article provides a summary of the 9th International Congress of the International Society for Pathophysiology (ISP 2023) which took place in Belgrade, Serbia, from 4 to 6 July 2023. It describes the main trends and the most promising areas of research in current pathophysiology, including investigations of new pathogenic pathways, and the identification of cellular and molecular mechanisms, target molecules, genetic mechanisms, and new therapeutic strategies. The present article also highlights the research conducted by leading scientific teams and national pathophysiological societies from various countries that adds to our understanding of the pathogenesis of diseases and pathological processes.

## Full-text entities

- **Genes:** TRAF2 (TNF receptor associated factor 2) [NCBI Gene 7186] {aka MGC:45012, RNF117, TRAP, TRAP3}, TNF (tumor necrosis factor) [NCBI Gene 7124] {aka DIF, IMD127, TNF-alpha, TNFA, TNFSF2, TNLG1F}, GNAI1 (G protein subunit alpha i1) [NCBI Gene 2770] {aka Gi, HG1B, NEDHISB}
- **Diseases:** diseases of the nervous system (MESH:D009422), injury to people or property (MESH:C000719191), necrotic (MESH:D009336), COVID-19 (MESH:D000086382), atherosclerotic (MESH:D050197), neurodegeneration (MESH:D019636), hyperhomocysteinemia (MESH:D020138), Alzheimer's disease (MESH:D000544), systemic inflammatory response syndrome (MESH:D018746), Inflammation (MESH:D007249), brain (MESH:D001927), autoimmune lesions of the endocrine glands (MESH:D004701), coronavirus infection (MESH:D018352), metabolic syndromes (MESH:D024821), metabolic diseases (MESH:D008659), shock (MESH:D012769), cardiomyopathy (MESH:D009202), Cancer (MESH:D009369), cardiac diseases (MESH:D006331), myocardial infarction (MESH:D009203), heart failure (MESH:D006333), autism (MESH:D001321), carcinogenesis (MESH:D063646), post-COVID-19 (MESH:D000094024), damage to the (MESH:D020263), cardiotoxic (MESH:D066126), neurological disorders (MESH:D009461), cardiovascular pathology (MESH:D002318), pulmonary hypertension (MESH:D006976), platelet function disorders (MESH:D001791)
- **Chemicals:** simvastatin (MESH:D019821), polyphenols (MESH:D059808), DOX (MESH:D004317), homocysteine (MESH:D006710), vitamin B6 (MESH:D025101), lipid (MESH:D008055), polyphenolic products (-)
- **Species:** Gammacoronavirus (genus) [taxon 694013], Homo sapiens (human, species) [taxon 9606]

## Full text

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## References

6 references — full list in the complete paper: https://tomesphere.com/paper/PMC10975917/full.md

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Source: https://tomesphere.com/paper/PMC10975917