Correction: A novel mechanism by which ACTA2-AS1 promotes cervical cancer progression: acting as a ceRNA of miR-143-3p to regulate SMAD3 expression
Lingli Luo, Min Wang, Xianping Li, Can Luo, Shan Tan, Sheng Yin, Lei Liu, Xiaolin Zhu
Abstract
Genes, proteins, chemicals, diseases, species, mutations and cell lines named across the full text — each resolved to its canonical identifier and authoritative record.
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Taxonomy
TopicsCancer-related molecular mechanisms research · Circular RNAs in diseases · RNA modifications and cancer
Correction: Cancer Cell Int (2020) 20:372 10.1186/s12935-020-01471-w
In this article [1], there was an error in Fig. 6. The corrected Fig. 6 is given below.Fig. 6. The efect of ACTA2-AS1/miR-143-3p/SMAD3 axis on progression of CC. a The knock-down ability of si-SMAD3 in HeLa and SiHa cells was confrmed by qRT-PCR. b The over-expression ability of pcDNA-ACTA2-AS1 was confrmed by qRT-PCR. c Cell viability was detected by CCK8 assay after transfecting with si-SMAD3 and co-transfecting with pcDNA‐ACTA2‐AS1 or miR-143-3p inhibitor. d The migration ability of transfected cells was analyzed by transwell assay. e Apoptosis condition of transfected cells was analyzed by fow cytometry analysis. ** P<0.05
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