Correction: Fission Yeast CSL Transcription Factors: Mapping Their Target Genes and Biological Roles
Martin Převorovský, Martina Oravcová, Jarmila Tvarůžková, Róbert Zach, Petr Folk, František Půta, Jürg Bähler
Abstract
Genes, proteins, chemicals, diseases, species, mutations and cell lines named across the full text — each resolved to its canonical identifier and authoritative record.
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Taxonomy
TopicsFungal and yeast genetics research · Biotin and Related Studies · RNA and protein synthesis mechanisms
After publication of this article [1], the authors discovered that one of the fission yeast strains used in the study had undergone a genome rearrangement, leading to artifactual results.
Here, the authors have provided additional information to clarify these issues:
The supposed Δcbf11 Δsty1 double deletion strain used in this article contains an ectopic (partial) copy of the cbf11 gene and, therefore, all experiments and conclusions based on this strain are invalid. The affected results appear in Fig 4 (all panels) of this article, and the invalid conclusion that the deletion of the stress-activated protein kinase Sty1 gene rescues the Δcbf11-associated defects is mentioned in the Abstract as well as throughout the Results and Discussion section. All other aspects and conclusions of our article remain valid. It is not possible to perform an analysis of a bona fide Δcbf11 Δsty1 strain to confirm that sty1 deletion does not suppress Δcbf11 because the Δcbf11 Δsty1 double mutant strain cannot be constructed because there seems to be synthetic lethality [2]. The validity of the other strains used in this article were analyzed and validated.
The authors have provided the following corrections:
In addition, the authors have provided an updated Fig 4 with the correct results and correct figure legend. Please see the complete, correct Fig 4 caption here:
The reference list from the paper itself. Each links out to its DOI / PubMed record.