# Hypoattenuated Leaflet Thickening-Associated Myocardial Infarction: A Rare Case Report and Literature Review

## TL;DR

A rare case shows that hypoattenuated leaflet thickening after a heart valve procedure may lead to a heart attack.

## Contribution

This case report highlights HALT as a potential cause of myocardial infarction in post-TAVR patients.

## Key findings

- An elderly patient on antiplatelet therapy experienced NSTEMI likely due to a thromboembolic event from HALT.
- HALT may contribute to myocardial infarction despite prophylactic antiplatelet use in post-TAVR patients.
- The case underscores the need to consider HALT-associated thromboembolism in post-TAVR MI diagnosis.

## Abstract

Hypoattenuated leaflet thickening (HALT), a potential aftereffect of transcatheter aortic valve replacement (TAVR) procedure, may affect valve performance and clinical outcomes. At this moment we describe an elderly patient who, despite being on prophylactic antiplatelet medication for previous percutaneous intervention (PCI) for coronary artery disease (CAD) and a self-expanding valve in-situ for aortic stenosis (TAVR), presented to the emergency room with non-ST-segment elevation myocardial infarction (NSTEMI), probably as a result of a thromboembolic event from HALT. The case highlights the significance of considering HALT-associated thromboembolism as a potential cause of myocardial infarction (MI) in post-TAVR patients.