Self-Organized Optical Pathways in Optofluidic Photonic Crystals

TL;DR

This study uses simulations to explore reconfigurable optofluidic pathways in silicon photonic crystals, revealing physical limits and control mechanisms for bio-inspired optical routing.

Contribution

It introduces a phenomenological feedback model for self-organized pathways and analyzes defect-mode dynamics in optofluidic photonic crystals.

Findings

Defect weakening causes faster transmission decay than bandgap narrowing.

Topology controls signal routing with high selectivity (S=0.98).

Self-organized pathways reach 63% of a hand-designed waveguide's transmission.

Abstract

This paper reports FDTD simulations of optofluidic reconfiguration in two-dimensional silicon photonic crystal waveguides, treating structural plasticity (the creation and destruction of optical pathways) via selective fluid infiltration. Using MPB eigenmode analysis, we decouple bandgap narrowing from defect-mode weakening, showing that defect weakening dominates (2.4 times faster transmission decay than bandgap narrowing at CS_2 indices). Infiltration topology controls signal routing (L-bend selectivity S = 0.98), though modulation depth is weak (Delta varepsilon/ varepsilon_ textSi = 11 %). A phenomenological optothermal feedback model produces self-organized pathways that achieve 63 % of a hand-designed waveguide's bandgap transmission (7.6 times the heavily suppressed empty-crystal baseline). Amplitude competition between counter-propagating sources produces strong, monotonic pathway steering (DeltaCOM_x from +0.03 to +4.92 ;a), while pulsed spike-timing-dependent plasticity yields a predictable null result: the timing-sensitive cross-term is suppressed by >10^2 when pulse delays exceed the temporal pulse width. The results provide benchmarks and identify physical limits for bio-inspired reconfigurable optofluidic photonics.