The lack of asymmetry of the Maxwell centroids, and of ocular dominance, in persons with dyslexia

TL;DR

This paper explores the lack of asymmetry in Maxwell centroids and ocular dominance in dyslexic individuals, linking it to interhemispheric connectivity issues and proposing non-invasive compensatory systems.

Contribution

It introduces a novel connection between Maxwell centroid symmetry and dyslexia, and suggests pulsed systems as effective compensation methods.

Findings

Lack of asymmetry correlates with dyslexia and reading difficulties.

Pulsed systems can instantly and non-invasively compensate for asymmetry.

Symmetrical and non-symmetrical connections induce confusions but are erasable.

Abstract

While the existence of an asymmetry between the two Maxwell centroids at the centre of the two foveas recorded using a foveascope, leads to the ocular dominance in good readers, the lack of asymmetry in most of the observers with dyslexia leads to their non-dominance and their difficulties in reading and writing. Indeed, the lack of asymmetry between the two main roads to the brain, i.e. the two optical nerves, leads to perturbations in the brain central connectivity, namely between the two hemispheres inducing too robust interhemispheric visual connections, beyond the critical period of 7-8 years. The symmetrical mirror-connections like b-d (observed for about 60% of children with dyslexia) or the non-symmetrical connections like b-b (35%) induce confusions and duplications in observers with dyslexia but remain erasable thanks to pulsed systems (glasses, lamps, screens. . .), using Hebbian processes in primary cortex synapses. The effect is instantaneous, non-invasive and compensates the lack of asymmetry. These systems help most of the observers with dyslexia to overcome their difficulties. The orthoptists and speech therapists can acquire and improve the mechanism to help the children to catch up, and perhaps be able to use the foveascope for an early diagnosis with young children.