Homophily and infections: static and dynamic effects
Matteo Bizzarri, Fabrizio Panebianco, Paolo Pin

TL;DR
This paper studies how homophily influences the spread of harmful states between groups with different immunization rates, revealing complex effects on steady states and infection dynamics depending on homophily levels and motivations.
Contribution
It provides a novel analysis of homophily's dual effects on infection diffusion and how motivations and endogenous immunization alter these impacts.
Findings
Homophily increases steady state infections when low, decreases when high.
Cumulative infections decrease with homophily at low levels, increase at high levels.
Group-specific motivations can make homophily harmful to both groups.
Abstract
We analyze the effect of homophily in the diffusion of a harmful state between two groups of agents that differ in immunization rates. Homophily has a very different impact on the steady state infection level (that is increasing in homophily when homophily is small, and decreasing when high), and on the cumulative number of infections generated by a deviation from the steady state (that, instead, is decreasing in homophily when homophily is small, and increasing when high). If immunization rates are endogenous, homophily has the opposite impact on the two groups. However, the sign of the group-level impact is the opposite if immunization is motivated by infection risk or by peer pressure. If motivations are group-specific, homophily can be harmful to both groups.
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Taxonomy
TopicsCOVID-19 epidemiological studies · Mathematical and Theoretical Epidemiology and Ecology Models · Influenza Virus Research Studies
