# Relation between winds and jets in radio-loud AGN

## TL;DR

This study reveals an inverse relationship between ionised wind density and jet strength in radio-loud AGN, suggesting a magnetic mechanism governs the outflow modes and indicating a fundamental link between winds and jets.

## Contribution

It provides the first systematic analysis linking wind properties to jet radio-loudness, proposing magnetic field configuration changes as the driving mechanism.

## Key findings

- Significant inverse correlation between wind column density and jet radio-loudness.
- Ionisation, inclination, and luminosity effects are unlikely causes.
- Evidence supports a wind-jet bimodality driven by magnetic field changes.

## Abstract

We investigate the relation between the two modes of outflow (wind and jet) in radio-loud active galactic nuclei (AGN). For this study we have carried out a systematic and homogeneous analysis of XMM-Newton spectra of a sample of 16 suitable radio-loud Seyfert-1 AGN. The ionised winds in these AGN are parameterised through high-resolution X-ray spectroscopy and photoionisation modelling. We discover a significant inverse correlation between the column density NH of the ionised wind and the radio-loudness parameter R of the jet. We explore different possible explanations for this NH-R relation and find that ionisation, inclination, and luminosity effects are unlikely to be responsible for the observed relation. We argue that the NH-R relation is rather a manifestation of the magnetic driving mechanism of the wind from the accretion disk. Change in the magnetic field configuration from toroidal to poloidal, powering either the wind or the jet mode of the outflow, is the most feasible explanation for the observed decline in the wind NH as the radio jet becomes stronger. Our findings provide evidence for a wind-jet bimodality in radio-loud AGN and shine new light on the link between these two modes of outflow. This has far-reaching consequences for the accretion disk structure and the wind ejection mechanism.